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bmp9  (Santa Cruz Biotechnology)


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    Santa Cruz Biotechnology bmp9
    Bmp9, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 93/100, based on 33 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/bmp9/product/Santa Cruz Biotechnology
    Average 93 stars, based on 33 article reviews
    bmp9 - by Bioz Stars, 2026-03
    93/100 stars

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    Fig. 8 Summary of the results. After the 10 weeks tilorone treatment, the HFD-induced increases in complex II-linked oxidative phosphorylation and com- plex IV activity reduced and the HFD-induced decreases in <t>BMP9,</t> pSmad1/5/8, and PPARγ levels were normal- ized. PET/MRI showed increased tissue uptake of 18FDG. As a consequence, lipid content of the liver decreased, the glycogen content increased, and blood glucose level, body mass, and liver and adipose tissue weights were normal- ized. HFD, high-fat diet; BMP, bone morphogenetic protein; PPARγ, peroxi- some proliferator-activated receptor gamma; 18FDG, 18F-fluoro-2-deoxyglucose
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    Fig. 8 Summary of the results. After the 10 weeks tilorone treatment, the HFD-induced increases in complex II-linked oxidative phosphorylation and com- plex IV activity reduced and the HFD-induced decreases in <t>BMP9,</t> pSmad1/5/8, and PPARγ levels were normal- ized. PET/MRI showed increased tissue uptake of 18FDG. As a consequence, lipid content of the liver decreased, the glycogen content increased, and blood glucose level, body mass, and liver and adipose tissue weights were normal- ized. HFD, high-fat diet; BMP, bone morphogenetic protein; PPARγ, peroxi- some proliferator-activated receptor gamma; 18FDG, 18F-fluoro-2-deoxyglucose
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    Fig. 8 Summary of the results. After the 10 weeks tilorone treatment, the HFD-induced increases in complex II-linked oxidative phosphorylation and com- plex IV activity reduced and the HFD-induced decreases in <t>BMP9,</t> pSmad1/5/8, and PPARγ levels were normal- ized. PET/MRI showed increased tissue uptake of 18FDG. As a consequence, lipid content of the liver decreased, the glycogen content increased, and blood glucose level, body mass, and liver and adipose tissue weights were normal- ized. HFD, high-fat diet; BMP, bone morphogenetic protein; PPARγ, peroxi- some proliferator-activated receptor gamma; 18FDG, 18F-fluoro-2-deoxyglucose
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    Fig. 8 Summary of the results. After the 10 weeks tilorone treatment, the HFD-induced increases in complex II-linked oxidative phosphorylation and com- plex IV activity reduced and the HFD-induced decreases in <t>BMP9,</t> pSmad1/5/8, and PPARγ levels were normal- ized. PET/MRI showed increased tissue uptake of 18FDG. As a consequence, lipid content of the liver decreased, the glycogen content increased, and blood glucose level, body mass, and liver and adipose tissue weights were normal- ized. HFD, high-fat diet; BMP, bone morphogenetic protein; PPARγ, peroxi- some proliferator-activated receptor gamma; 18FDG, 18F-fluoro-2-deoxyglucose
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    Fig. 8 Summary of the results. After the 10 weeks tilorone treatment, the HFD-induced increases in complex II-linked oxidative phosphorylation and com- plex IV activity reduced and the HFD-induced decreases in <t>BMP9,</t> pSmad1/5/8, and PPARγ levels were normal- ized. PET/MRI showed increased tissue uptake of 18FDG. As a consequence, lipid content of the liver decreased, the glycogen content increased, and blood glucose level, body mass, and liver and adipose tissue weights were normal- ized. HFD, high-fat diet; BMP, bone morphogenetic protein; PPARγ, peroxi- some proliferator-activated receptor gamma; 18FDG, 18F-fluoro-2-deoxyglucose
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    Fig. 8 Summary of the results. After the 10 weeks tilorone treatment, the HFD-induced increases in complex II-linked oxidative phosphorylation and com- plex IV activity reduced and the HFD-induced decreases in BMP9, pSmad1/5/8, and PPARγ levels were normal- ized. PET/MRI showed increased tissue uptake of 18FDG. As a consequence, lipid content of the liver decreased, the glycogen content increased, and blood glucose level, body mass, and liver and adipose tissue weights were normal- ized. HFD, high-fat diet; BMP, bone morphogenetic protein; PPARγ, peroxi- some proliferator-activated receptor gamma; 18FDG, 18F-fluoro-2-deoxyglucose

    Journal: GeroScience

    Article Title: Tilorone attenuates high-fat diet-induced hepatic steatosis by enhancing BMP9-Smad1/5/8 signaling.

    doi: 10.1007/s11357-025-01685-8

    Figure Lengend Snippet: Fig. 8 Summary of the results. After the 10 weeks tilorone treatment, the HFD-induced increases in complex II-linked oxidative phosphorylation and com- plex IV activity reduced and the HFD-induced decreases in BMP9, pSmad1/5/8, and PPARγ levels were normal- ized. PET/MRI showed increased tissue uptake of 18FDG. As a consequence, lipid content of the liver decreased, the glycogen content increased, and blood glucose level, body mass, and liver and adipose tissue weights were normal- ized. HFD, high-fat diet; BMP, bone morphogenetic protein; PPARγ, peroxi- some proliferator-activated receptor gamma; 18FDG, 18F-fluoro-2-deoxyglucose

    Article Snippet: Then, the membranes were blocked in Tris buffered saline containing 5% skimmed milk and 0.1% Tween-20 (Sigma-Aldrich) for 1 h at room temperature and were incubated at 4 °C overnight with the following rabbit polyclonal primary antibodies: phospho-Smad1/5/8 (Smad1 [Ser463/465]/Smad5 [Ser463/465]/Smad9 [Ser465/467]; #AB-3848-J; Sigma-Aldrich); Smad1/5/8 (#56,656; Novus Biologicals; Centennial, CO, USA); PPARγ (#2435; Cell Signaling Technology; Danvers; MA; USA); PGC-1α (#2178; Cell Signaling Technology); TOM20 (#42,406; Cell Signaling Technology); BMP6 (#55,421–1-AP; Proteintech; Rosemont; IL, USA); BMP9 (#17,769–1-AP; Proteintech); and GAPDH (#2188; Cell Signaling Technology).

    Techniques: Phospho-proteomics, Activity Assay